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EBNA-3a seems to regulate a subset of mobile genes, many of which overlap with Those people regulated by EBNA2. The two proteins can act antagonistically or cooperatively to manage gene expression. These results place EBNA-3a like a crucial modulator of B mobile proliferation and survival for the duration of EBV latency, and for a pertinent CD8⁺ T mobile focus on in equally simple immunological scientific studies and translational analysis.
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